I’ve tested out many kinds of diet which lasted no more than one month. My stomach has met more vegetables than a traditional Holland glasshouse is able to produce in 100 years. And do you think I was successful? If flatulency is name for weight loss I had been very successful young man. Unfortunately gas-escape evokes just cubage loss but not weight loss. I’m punished by my own genes, what a betrayal. Hunger is my shadow and try to get rid of your own shadow.

Yesterday night for instance. I could not fall asleep because I was pretty hungry, briliant. I offered two choices to myself.

 

1) In the face of relatively heavy tiredness, reach the fridge a eat something light. (It was clear right at the beginning that if I chose this way I would eat half an hour without break. Of course the offer does not include this. But just until I make decision.) And result? Just a little bit of fat in addition.

2) Close your eyes and attempt to fall asleep. You will suffer, fall asleep very late and maybe hunger will wake you up and the quality of sleep be like something of poor quality.

I said good bye to obesity couple years ago. But weight loss is only the first step. The next steps should be following months and years full of discipline. I managed this because I realized that there is no way without deep knowledge of human metabolism. I tried to provide you the essential knowledge of how the metabolism of obese people works and how does it differ from metabolism of slim people. If you really understand the rules of human metabolism you will get the absurdity of most of diets which seem to be made up by specialists but mostly it is just a commercial trick. Human being is an individuality and every one of us knows himself much more than anyone else. Everyone needs something different. We need knowledge to know what our body needs.

Amount of fat depositions must be regulated and this is task for leptin. In the process of getting thicker fat cells start to enlarge themselves and produce much more leptin which comes into brain by blood and gives a signal : there is too much fat. Leptin receptors react and two changes happen: Decreasing of hunger and increase of energy for metabolism and human activity.

The more fat increasing the more leptin incoming into blood. In the properly functional metabolism the higher level of leptin in blood causes energy intake so our body would have enough energy for activity and following energy expenditure. In the case of metabolic imperfection the fat cells produce enough leptin but in brain there is not enough amount of active leptin receptors for incoming signals and therefore metabolism is not stimulated enough to expenditure of energy from consumed fat. The point is the majority of chemical signals is wasted because of not enough receptors and brain is confused and deaf to rest of the leptin signals. This is leptin resistance.

Leptin resistance is a result of two reasons. The first one is already mentioned genes predisposition. The second one is inhibition of leptin receptors caused by wrong eating manners. For example, starving whole day and big overeating in the evening. Overeating evokes fat cells to be persuaded they haven’t produced enough leptin and start to produce much more. Increased concentration of leptin in blood supercharge our brain which starts with inhibition certain amount of receptors what makes worse the problem. The result is brain deafness to leptin because of receptors inhibition, disturbance of this correlation and inhibition of whole metabolism.Brain doesn’t feel power of signals, doesn’t calm hunger down and doesn’t realize we consumed too much fat and energy and doesn’t turn it into promtly available energy. Result? Fat storing, tiredness, mental lazyness and permanent hunger. So … eat small portions but more often. It last some time till blood transports leptin to receptors and till brain signalizes satiety.

With insulin resistance blood is filled by sugar to certain level and muscle cells receptors do not respond enough to insulin. So in blood there is a lot of insulin which should get sugar into muscle cells but insulin receptors are deaf to insulin. It causes that the blood level stays higher and body is persuaded it haven’t produced enough insulin and pancreas goes on with producing. As a result there is a much more sugar and insulin in blood for one or two hours. And what about the fat metabolism? Because blood is full of sugar and energy, body has no need to turn fat into energy as a alternative.

That’s why ( insulin resistance person) the process of fat burning is stopped after eating carbohydrate with high glycemic index. It a reason why you should eat fat sweet desserts only you are under death threaten. If your body has a tendency to fat storing you will get into a vicious circle: the more fat you’ve got the stronger is your insulin resistance and you’re getting thicker and thicker.

What is the reason? Inheritance (omg why me?) or the similar reason like leptin resistance does. Low-calories diets or if food contains high percentage of fat cause insulin receptors amount reduction. If the insulin receptors of muscle cells stop react to insulin and do not allow sugar to get into the cells, the pancreas produces more insulin. Instead of muscle cells receptors begin to react to increasing amount of insulin, the situation is getting worse. If this process reached certain state it would be spoken about diabetes.

Main task in treatment for insulin resistance is to reduce percentage of fat of our body and our food. Because of the vicious circle I spoke about the fatter we are the stronger our insulin resistance is.

In woman body estrogen is important too in relation with insulin resistance. Estrogen influences insulin resistance. If your estrogen level is high your insulin resistance will be stronger and you will have tendency to obesity. The more fat the more production of estrogen and it is a new version of visious circle.

During making of alcohol (except of destillate) the yeast fungus change the sugar into alcohol and it suck the energy from the alcohol then. In liver there is a opposite process. 50% energy of beer is the energy from sugar. Destillate contains a little bit of sugar (except of rum which contains the same amount of sugar as wine does), it is only ethanol,water and some volatile matter.

Sugar does not accumulate in the body and does not turn into fat but turns into energy. The rest of the energy from alcohol is energy from ethanol which is used to oxidation of ethanol (liquidation) and is used for thermoregulation. Metabolism is intensified because there is demand of more energy for oxidation of ethanol . In other words for liquidation of ethanol there have to be enough energy for this liquidation. This is in connection with the sudden warming-up during drinking. What’s most important, alcohol is a toxin and if the liver did not get rid of this toxin it would have death consequences.

If we consume a big amount of alcohol it makes liver overloaded because it must get rid of it and that’s why liver can’t do the common activities like to supply blood with sugar. If we do not eat for long time the liver has to supply blood with sugar to certain level no matter whether the source is resources of fat or proteins. But in the case liver must do liquidation of ethanol, it can’t do this job. And the consequences? Not enough sugar in blood and minimal fat burning. If it to be a contrary it does not work even turning fat into energy if it was consumed in the time of drinking alcohol. So there is a tendency to storing fat in the shortest way.